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Long-COVID, A Post-Infectious Autoimmune Syndrome: Understanding Commonalities and Biological Clues from Other Post-Infectious Autoimmune Syndromes
Presentation and talk with Craig Shimasaki, PhD, MBA, Cofounder & CEO, Moleculera Labs

In this presentation we will discuss biological, molecular, and clinical data explaining how molecular mimicry, through infections such as Strep, Lyme, and now SARS-CoV-2, can trigger immune dysregulation including autoantibodies directed against targets in the brain that interrupt normal neurologic functions.

Long-COVID is understood to be a condition where individuals experience a variety of neurologic, psychiatric, cardiovascular, and pulmonary symptoms many months after initially contracting the SARS-CoV-2 virus. Other post-infectious autoimmune syndromes may help us understand Long-COVID such as, persistent neurologic Lyme, Pediatric Acute Onset Neuropsychiatric Syndrome (PANS), Autoimmune Encephalopathy (AE), Basal Ganglia Encephalitis (BGE), and others.

Our understanding of Long-COVID is rapidly evolving. Infection-triggered autoimmune syndromes and molecular mimicry are medical model that may help us understand the pathophysiology of Long-COVD and lead to the integration of new diagnostics and more effective therapies to treat this rapidly expanding disorder.

Apr 7, 2021 07:00 PM in London

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